DERMATITIS HERPETIFORME PDF

Dermatitis herpetiformis DH is a chronic autoimmune blistering skin condition, [3] characterised by blisters filled with a watery fluid [4] that is intensely itchy. DH is a cutaneous manifestation of Coeliac disease. Dermatitis herpetiformis was first described by Louis Adolphus Duhring in The age of onset is usually about 15—40, but DH also may affect children and the elderly.

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Dermatitis herpetiforme is a chronic, hereditary disease having an immune basis. It preferentially affects the Northern Europe population. Its approximate frequency is one case per every ten thousand persons and it predominates in males with a 1.

At present, it is considered to be a manifestation form of celiac disease CD , the participation of gluten being clearly demonstrated in its development.

Clinically, it is characterized by vesicle-bullous type skin lesions, accompanied by intense itching. Increase of the serological markers existing in CD has been manifested, although the only way of establishing the definite diagnosis is by demonstration of granular IgA deposits in the dermoepidermic junction of healthy or perilesional skin.

In all the cases, the intestinal mucosa is involved, which is verified by a duodenal biopsy. Although dapsone has been shown to be effective for symptomatic treatment, initiation of a gluten free diet for all one's life is essential.. Inicio Medicina de Familia. ISSN: Descargar PDF.

Autor para correspondencia. Unidad Docente. Palabras clave:. Although dapsone has been shown to be effective for symptomatic treatment, initiation of a gluten free diet for all one's life is essential. Key words:. JAMA, 3 , pp. Dermatitis herpetiformis: coeliac disease of the skin. Ann Med, 30 , pp.

Gawkrodger, J. Blackwell, H. Dermatitis herpetiformis: diagnosis, diet and dermography. Gut, 25 , pp. Reunala, J. Acta Dermato-Venereol, 58 , pp. Mobracken, W. Kastrup, L. Incidence and prevalence of dermatitis herpetiformis in western Sweden. Acta Dermato-Venereol, 64 , pp. Incidence and prevalence of dermatitis herpetiformis in a country in central Sweden, with comments on the course of the disease and IgA deposits as diagnostic criterion. Christensen, M. Hindsen, A. Natural history of dermatitis herpetiformis in southern Sweden.

Smith, J. Tulloch, L. Meyer, J. The incidence and prevalence of dermatitis herpetiformis in Utah. Arch Dermatol, , pp. Fry, P. Keir, R. Small intestinal structure and function, and haematological changes in dermatitis herpetiformis. Lancet, ii , pp. Fry, R. McMinn, J. Cowan, A. Effect of glutenfree diet on dermatological, intestinal and haematological manifestations of dermatitis herpetiformis.

Lancet, i , pp. Gluten-free diet and reintroduction of gluten in dermatitis herpetiformis. Seath, D. Riches, A. Clearence of skin lesions in dermatitis herpetiformis after gluten withdrawal. Fry, J. Leonard, A. Long term follow up of dermatitis herpetiformis with and without dietary gluten withdrawal. Br J Dermatol, , pp. Greenberg, S. Hodge, J. Evidence for recessive and against dominant inheritance at the HLA linked locus in coeliac disease.

Am J Human Gen, 34 , pp. Reunala, S. Clin Dermatol, 9 , pp. Reunala, O. Salo, P. Ann Clin Res, 8 , pp. Balas, J. Vicario, A. Zambrano, D. Acuna, D. Tissue Antigens, 50 , pp. Seah, P. The small intestine in dermatitis herpetiformis. J Clin Pathol, 27 , pp. Hall, K. Comparison of the intestinal and serum antibody response in patients with dermatitis herpetiformis.

Clin Inmunol Inmunopathol, , pp. Graeber, B. Baker, J. The role of cytokines in the generation of skin lesions in dermatitis herpetiformis. Savilahti, T. Ormala, A. Arato, G. Hacsek, K. Holm, T. Klemola, et al. Clin Exp Immunol, , pp. Identification of tissue transglutaminase as the autoantigen of celiac disease. Nature Med, 3 , pp. Garioch, H. Lewis, S. Sargent, J. Leonard, L. Cuartero, M. Santamaria, M. Acuna Quiros, J. Romero Portilla, C. Pedron Giner, et al. Dermatitis herpetiformis vs.

An Exp Pediatric, 37 , pp. Demoulins-Giacco, V.

PELAGICEV NARODNI UCITELJ PDF

Dermatitis herpetiformis

Dermatitis herpetiformis. International Journal of Dermatology ; Oxentenko A, Murray J. Celiac disease and dermatitis herpetiformis: the spectrum of gluten-sensitive enteropathy. Epidermal transglutaminase TGase 3 is the autoantigen of dermatitis herpetiformis.

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Dermatitis herpetiforme is a chronic, hereditary disease having an immune basis. It preferentially affects the Northern Europe population. Its approximate frequency is one case per every ten thousand persons and it predominates in males with a 1. At present, it is considered to be a manifestation form of celiac disease CD , the participation of gluten being clearly demonstrated in its development. Clinically, it is characterized by vesicle-bullous type skin lesions, accompanied by intense itching. Increase of the serological markers existing in CD has been manifested, although the only way of establishing the definite diagnosis is by demonstration of granular IgA deposits in the dermoepidermic junction of healthy or perilesional skin. In all the cases, the intestinal mucosa is involved, which is verified by a duodenal biopsy.

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